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NDT Advance Access published online on November 24, 2006
Nephrology Dialysis Transplantation, doi:10.1093/ndt/gfl702
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© The Author [2006]. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

Primary hyperaldosteronism in a patient with end-stage renal disease

Amir Kazory1 and I. David Weiner1,2

1Division of Nephrology, Hypertension and Transplantation, University of Florida and 2Nephrology and Hypertension Section, North Florida/South Georgia Veterans Health System, Gainesville, FL 32610, USA

Correspondence and offprint requests to: Correspondence and offprint requests to: Amir Kazory, MD, Division of Nephrology, University of Florida, 1600 SW Archer Rd, Gainesville, FL 32610, USA. Email: amir.kazory@medicine.ufl.edu

Keywords: aldosterone; end-stage renal disease; haemodialysis; hypertension

The first 150 words of the full text of this article appear below.



   Introduction
 
The prevalence of hypertension in patients on chronic haemodialysis has been reported to be as high as 90% [1]. Several aetiological factors may contribute to hypertension in this population including sodium and water retention, increased activity of vasoconstrictive systems and increased arterial stiffness [2]. Recent studies show that primary aldosteronism is common in patients with hypertension, being present in up to 10% of all hypertensive individuals [3]. Our understanding of the physiological and pathophysiological actions of aldosterone has changed in recent years, and increasing evidence identifies that its role in the pathogenesis of hypertension is beyond its effects on renal tubular cell-mediated ion transport and NaCl retention. Here, we present a case of hypertension secondary to primary aldosteronism in an anuric patient on chronic haemodialysis who showed a dramatic response to aldosterone receptor blockade.



   Case
 
A 57-year-old African American male with a history of progressive . . . [Full Text of this Article]



   Discussion
 

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