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NDT Advance Access originally published online on March 14, 2008
Nephrology Dialysis Transplantation 2008 23(6):2081-2083; doi:10.1093/ndt/gfn097
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© The Author [2008]. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved. For Permissions, please e-mail: journals.permissions@oxfordjournals.org


Hyperkalaemia as a complication of ureteroileostomy: a case report and literature review

Nizar Eskandar and Jean L. Holley

Nephrology Division, University of Virginia Health System, Box 800133, Charlottesville, VA 22908, USA

Correspondence and offprint requests to: Nizar Eskandar, Division of Nephrology, University of Virginia Health System, Box 800133, Charlottesville, VA 22908, USA. Tel: +1-434-924-5125; Fax: +1-434-924-5848; E-mail: ne9u@virginia.edu

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   Introduction
 
Ureteral diversion in which the ureter is implanted into either the sigmoid colon or a short loop of ileum is associated with multiple metabolic complications [1,2]. Ureterosigmoidostomy commonly leads to metabolic acidosis due to the presence of colonic anion exchange pumps that reabsorb luminal chloride as bicarbonate is secreted across the sigmoid colon. In such cases, reabsorption of urinary ammonium that contacts the sigmoid colon may also contribute to metabolic acidosis [1]. In addition, sigmoid loops usually lead to hypokalaemia due to colonic potassium secretion. However, if the intestinal conduit in contact with ureteral contents is jejunum, hyperkalaemia may occur, presumably due to absorption of urinary potassium by the jejunum. This infrequent . . . [Full Text of this Article]



   Case
 


   Discussion
 


   Summary
 

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