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NDT Advance Access originally published online on December 29, 2005
Nephrology Dialysis Transplantation 2006 21(4):1100-1103; doi:10.1093/ndt/gfk019
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© The Author [2005]. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

Case Report

Hepatic candidiasis in a kidney transplant recipient treated successfully with amphotericin B and itraconazole

Cesar Yaghi1, Aida Moussali2, Gerard Abadjian3, Bahaa Kheir1, Lina Menassa4, Rita Slim1, Khalil Honein1 and Raymond Sayegh1

1 Departments of Gastroenterology, 2 Nephrology, 3 Clinical Pathology and 4 Radiology, Hotel Dieu de France, Beirut, Lebanon

Correspondence and offprint requests to: Cesar Yaghi, Department of Gastroenterology, Hotel Dieu de France, Beirut, Lebanon. Email: cesar.yaghi@usj.edu.lb

Keywords: Candida; hepatic candidiasis; kidney; liver; mycosis; transplantation

The first 10% of the full text of this article appears below.

The incidence of hepatic candidiasis is difficult to estimate because of diagnostic difficulties. Its frequency was ~7% in a study of 562 adult patients with leukaemia [1]. Hepatic candidiasis (HC), also referred to as chronic disseminated candidiasis, hepatosplenic candidiasis and granulomatous hepatic candidiasis, affects almost exclusively patients undergoing remission induction chemotherapy or bone marrow transplantation for acute leukaemia [2]. It occurs on recovery following prolonged episodes of bone marrow dysfunction and neutropenia [1]. The liver, spleen and sometimes the kidneys are infected with Candida. Occasionally, patients with other types of immunosuppression (aplastic anaemia, lymphoma, sarcoma or liver transplantation) may develop hepatosplenic candidiasis [3]. This is the first reported case of HC in a kidney transplant recipient.



   Case
 
A 37-year-old woman had cadaveric kidney transplantation with five human leukocyte antigen (HLA) compatibilities, . . . [Full Text of this Article]



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