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NDT Advance Access originally published online on August 5, 2006
Nephrology Dialysis Transplantation 2006 21(10):2953-2956; doi:10.1093/ndt/gfl197
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© The Author [2006]. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

Albuminuria in acute tubular necrosis

Bradford Lee West1, Maria M. Picken2 and David J. Leehey1,

1Department of Medicine and 2Department of Pathology, Loyola University Medical Center, Maywood, Illinois, USA

Correspondence and offprint requests to: David J. Leehey, MD, Loyola University Medical Center, Bldg 102, Rm 3661, 2160 S 1st Ave, Maywood, IL 60153, USA. Email: dleehey@lumc.edu

Keywords: acute tubular necrosis; albuminuria; endocytosis

The first 150 words of the full text of this article appear below.



   Introduction
 
Acute renal failure (ARF) is defined as an abrupt decline in the glomerular filtration rate (GFR), as evidenced by rising serum levels of urea nitrogen and creatinine. Acute tubular necrosis (ATN) is the most common cause of ARF. ATN is an acute decline in glomerular filtration as a result of damage to the tubules in the kidney. Sepsis is the most common aetiology of ATN, causing ARF in 20–25% of normotensive patients and 50% of hypotensive patients [1].

The clinical diagnosis of ATN can be difficult as there are no uniform diagnostic criteria. Typical findings suggesting ATN are muddy brown granular or tubular epithelial cell casts, fractional excretion of sodium (FeNa) greater than 1%, and isosthenuria. ATN is not expected to affect the glomerulus, and thus should not cause glomerular proteinuria [2,3].

In this report, we describe a young woman with ARF and albuminuria . . . [Full Text of this Article]



   Case report
 


   Discussion
 

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