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NDT Advance Access originally published online on May 17, 2005
Nephrology Dialysis Transplantation 2005 20(7):1515-1516; doi:10.1093/ndt/gfh886
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© The Author [2005]. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved. For Permissions, please email: journals.permissions@oupjournals.org

Letter

High clotting factor activities and enhanced platelet-related haemostasis in a patient with recurrent episodes of thrombotic–thrombocytopenic purpura: additional risk factors for onset and recurrence?

The first 10% of the full text of this article appears below.

Sir,

Thrombotic–thrombocytopenic purpura (TTP) is a life-threatening disorder characterized by microvascular thrombi, mainly in arterioles and capillaries, low platelet count, haemolysis and neurological deficits [1]. Impaired activity of the von Willebrand factor (vWF) cleaving protease ADAMTS 13, leading to the accumulation of ultralarge vWF multimers, was found to play a major role in the pathogenesis of TTP [2]. Consequently, plasma exchange became the therapy of choice supplying . . . [Full Text of this Article]

Christoph Sucker1, Firuseh Farokhzad2, Christine Kurschat2, Bernd Grabensee2, Marcus Stockschlaeder1, Rainer B. Zotz1 and Rudiger E. Scharf1

1 Department of Hemostasis and Transfusion Medicine2 Department of Nephrology Heinrich Heine University Medical Center Duesseldorf Germany Email: sucker@med.uni-duesseldorf.de


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