Skip Navigation

This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (1)
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Segawa-Takaeda, C.
Right arrow Articles by Yokoyama, H.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Segawa-Takaeda, C.
Right arrow Articles by Yokoyama, H.
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Nephrol Dial Transplant (2002) 17: 172-174
© 2002 European Renal Association-European Dialysis and Transplant Association


Letters

Focal glomerulosclerosis expanding from the glomerular vascular pole in a Japanese male with mitochondrial-DNA mutation

Chikako Segawa-Takaeda1, Shin-ichi Takeda1, Yasuhiko Ieki1, Eisuke Takazakura1, Joji Haratake2, Takashi Wada3 and Hitoshi Yokoyama3

1 Internal Medicine 2 Pathology of Kurobe City Hospital, Kurobe, Toyama 3 First Department of Internal Medicine and Division of Blood Purification, Kanazawa University School of Medicine Kanazawa, Ishikawa, Japan

Sir,

Recently mitochondrial DNA mutations have been detected in patients with various diseases, in particular, mitochondrial encephalomyopathies. With regard to renal involvement, renal tubular dysfunction resulting in renal tubular acidosis and de-Toni-Debre-Fanconi syndrome were reported for the first time in 1977 [1]. In addition, recent studies reveal that focal glomerulosclerosis (FGS) and chronic renal failure are accompanied by mitochondrial abnormality [2–5]. Little is known, however, about the mechanism and progression of glomerular injury induced by mitochondrial mutations. We report a case of . . . [Full Text of this Article]

References


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?