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Nephrol Dial Transplant (2000) 15: 1290-1292
© 2000 European Renal Association-European Dialysis and Transplant Association

Editorial Comments

Proteinuria in the transplanted patient

Alexander R. Rosenkranz and Gert Mayer

Leopold Franzens Universität Innsbruck, Universitätsklinik für Innere Medizin, Klinische Abteilung für Nephrologie, Innsbruck, Austria

Proteinuria and progressive renal disease—the unfortunate twins

The phenomenon of glomerular proteinuria has received considerable attention since it was suggested by Remuzzi and co-workers [1] that increased urinary protein concentrations might per se contribute to the progressive nature of renal disease. Findings of the Ramipril Efficacy in Nephropathy (REIN) studies are compatible with this assumption as the degree of nephroprotection obtained by angiotensin-converting enzyme (ACE) inhibitor therapy much better correlated with the reduction of urinary protein excretion than with systemic haemodynamic effects [2].

Although a definite pathophysiological link between proteinuria and progression of renal disease has yet to be established, it is generally accepted that proteinuria is at least a reliable prognostic marker. This is true for patients with secondary focal and segmental glomerulosclerosis, the structural correlate typically present in patients with a reduction of nephron mass [3], but also at least in some ‘immunologically’ mediated primary glomerulonephritides. . . . [Full Text of this Article]

Is interstitial allograft disease after transplantation mediated by urinary proteins?

Will treatment of post-transplant proteinuria preserve renal allograft function?

Notes

References


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