Skip Navigation

This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (4)
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Frey, F. J.
Right arrow Articles by Ferrari, P.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Frey, F. J.
Right arrow Articles by Ferrari, P.
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Nephrol Dial Transplant (2000) 15: 1512-1514
© 2000 European Renal Association-European Dialysis and Transplant Association


Editorial Comments

Pastis and hypertension—what is the molecular basis?

Felix J. Frey and Paolo Ferrari

Division of Nephrology/Hypertension, University Hospital of Berne, Berne, Switzerland

Glycyrrhiza glabra

The therapeutic properties of Glycyrrhiza glabra were already known by Egyptians, Greeks, and Romans in antiquity [1]. They used extracts from this plant for a diversity of ailments and as a sweetener. In the modern society it is found in drinks such as Belgian beers, Ouzo, Pernod or Pastis brands. Many chewing gums contain glycyrrhetinic acid. The rationale for adding glycyrrhetinic acid, the active ingredient of liquorice, to chewing gums is the observation that, contrary to glucose, liquorice does not promote bacterial growth and adherence of cariogenic bacteria [2]. In addition liquorice is often added to confectionery. The discovery of the value of liquorice — previously marketed as carbenoxolone, an oleandane derivative of glycyrrhetinic acid — in the treatment of peptic ulcer allowed researchers to establish its adverse effect on salt and water metabolism.

Clinical features and erroneous interpretation

Patients with excessive ingestion of liquorice present with hypokalaemic . . . [Full Text of this Article]

Mechanism of renal sodium retention and potassium loss induced by liquorice

11ß-HSD isoenzymes

Health hazards of liquorice

Acknowledgments

Notes

References


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
HypertensionHome page
I. Quack, O. Vonend, L. Sellin, J. Stegbauer, G. Dekomien, and L. C. Rump
A Tale of Two Patients With Mendelian Hypertension
Hypertension, March 1, 2008; 51(3): 609 - 614.
[Full Text] [PDF]


Home page
Nephrol Dial TransplantHome page
A. Woywodt, A. Herrmann, H. Haller, and M. Haubitz
Severe hypokalaemia: is one reason enough?
Nephrol. Dial. Transplant., November 1, 2004; 19(11): 2914 - 2917.
[Full Text] [PDF]


Home page
Nephrol Dial TransplantHome page
M. G. Mohaupt and F. J. Frey
Mineralocorticoid receptor malfunction: further insights from rare forms of hypertension
Nephrol. Dial. Transplant., April 1, 2002; 17(4): 539 - 542.
[Full Text] [PDF]


Home page
Nephrol Dial TransplantHome page
F. J. Frey
The hypertensive patient with hypokalaemia: the search for hyperaldosteronism
Nephrol. Dial. Transplant., June 1, 2001; 16(6): 1112 - 1116.
[Full Text] [PDF]