Skip Navigation

This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (2)
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Naito, T.
Right arrow Articles by Nihei, H.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Naito, T.
Right arrow Articles by Nihei, H.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Nephrol Dial Transplant (1999) 14: 2716-2719
© 1999 European Renal Association-European Dialysis and Transplant Association

Case Reports

Progressive tumoral calcinosis as the presenting feature of sarcoidosis in a patient on haemodialysis treatment

Takashi Naito1, Kosaku Nitta1, Naoki Kimata1, Kazuho Honda1, Takumi Yoshida1, Miyuki Koinuma1, Yoko Ikeda1, Yoshiharu Kato2 and Hiroshi Nihei1

1 Department of Medicine, Kidney Centre and 2 Department of Orthopedics, Tokyo Women's Medical University, Tokyo, Japan

Correspondence and offprint requests to: K. Nitta MD, Department of Medicine, Kidney Centre, Tokyo Women's Medical University, 8-1 Kawada-cho, Shinjuku-ku, Tokyo 162-8666, Japan.

Keywords: etidronate disodium; haemodialysis; sarcoidosis; tumoral calcinosis



   Introduction
 
Periarticular tumoral calcification is an infrequent, but severe, complication in uraemic patients. The genesis of tumoral calcification has generally been explained by a high calcium–phosphorus product (CaxP) and/or advanced secondary hyperparathyroidism after long-term haemodialysis (HD) treatment [1]. We observed a male patient who developed rapidly progressive tumoral calcinosis less than 1 year after the beginning of HD. He had sarcoidosis and a high CaxP product, but not severe hyperparathyroidism.



   Case
 
A 39-year-old man was admitted in September 1995, because of appetite loss and general fatigue. Physical examination revealed marked uraemia, and HD was started three times a week on 26 September. However, bilateral ankle pain appeared immediately from the beginning of HD and the ankles gradually developed swelling and hyperaemia. Right wrist pain also developed in April . . . [Full Text of this Article]



   Discussion
 


   References
 

Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?