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NDT Advance Access originally published online on December 13, 2007
Nephrology Dialysis Transplantation 2008 23(4):1107-1108; doi:10.1093/ndt/gfm867
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© The Author [2007]. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved. For Permissions, please e-mail: journals.permissions@oxfordjournals.org


T-cells in angiotensin-II-induced vascular damage*

Geiger Helmut

Division of Nephrology, Department of Internal Medicine, Johann Wolfgang Goethe-University Frankfurt/Main, Theodor-Stern Kai 7, 60590 Frankfurt, Germany

Helmut Geiger, Division of Nephrology, Department of Internal Medicine, Johann Wolfgang Goethe-University Frankfurt/Main, Theodor-Stern Kai 7, 60590 Frankfurt, Germany. E-mail: h.geiger@em.uni-frankfurt.de

Keywords: hypertension; T-cells

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In this study, Guzik et al. proved that mice lacking T- and B-cells do not develop hypertension and associated vascular dysfunction in two different models of hypertension, either with high angiotensin II (angiotensin II infusion) or with low angiotensin II (DOCA salt). Transfusion of T, but not B, cells restored these abnormalities, leading to the conclusion that T-cells play an important role in the genesis of hypertension. To gain an insight into how T-cells may be involved in the mechanisms that finally cause hypertension, they examined the effects of angiotensin II on vascular infiltration of these . . . [Full Text of this Article]


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