Skip Navigation


NDT Advance Access originally published online on January 26, 2008
Nephrology Dialysis Transplantation 2008 23(4):1101-1102; doi:10.1093/ndt/gfm933
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow All Versions of this Article:
23/4/1101    most recent
gfm933v1
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Gross, P.
Right arrow Articles by Meye, C.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Gross, P.
Right arrow Articles by Meye, C.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© The Author [2008]. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved. For Permissions, please e-mail: journals.permissions@oxfordjournals.org


Proximal RTA: Are all the charts completed yet?

Peter Gross and Constanze Meye

Division of Nephrology, Department of Medicine III, University Medical Center C. G. Carus, Dresden, Germany

Professor Peter Gross, M.D., Division of Nephrology, Department of Medicine III, Universitats Klinikum C. G. Carus Fetscherstrasse 74 01307 Dresden, Germany, Fax: 0049 351 458 5333, Tel: 0049 351 458 2645, E-mail: Peter.Gross@uniklinikum-dresden.de

Keywords: metabolic acidosis; renal tubular acidosis; tubular transport proteins

The first 10% of the full text of this article appears below.

Renal tubular acidosis (RTA) is an uncommon disorder; however, the subgroup of isolated familial proximal RTA (pRTA) is exceedingly rare. The term ‘isolated’ pRTA distinguishes these disorders from Fanconi syndrome, in which in addition to acid excretion, other proximal tubular functions are impaired as well. In this issue of Nephrology Dialysis Transplantation, Katzir et al. [1] report findings in a single family with a specific form of isolated familial pRTA. It is highly likely that only very few physicians will ever encounter a case of this nature in their professional lives. Why then would this report be of interest?

The human body generates ~50–100 mmol of . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?