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NDT Advance Access originally published online on October 13, 2008
Nephrology Dialysis Transplantation 2008 23(12):3743-3745; doi:10.1093/ndt/gfn561
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© The Author [2008]. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved. For Permissions, please e-mail: journals.permissions@oxfordjournals.org


A little help from our friends: what an epidemiologic study teaches us about autoinflammation, granuloma and proteinase-3-specific antineutrophil cytoplasmic autoantibodies

Peter Lamprecht and Wolfgang L. Gross

University of Lübeck, Department of Rheumatology, Vasculitis Center UKSH & Rheumaklinik Bad Bramstedt, Ratzeburger Allee 160, 23538 Lübeck, Germany

Correspondence and offprint requests to: Peter Lamprecht, MD, University of Luebeck, Vasculitis Center UKSH & Rheumaklinik Bad Bramstedt, Ratzeburger Allee 160, 23538 Luebeck, Germany. Tel: +0049-451-500-2368; Fax: +0049-451-500-3650; E-mail: peter.lamprecht@rheuma.uni-luebeck.de

Keywords: ANCA; autoinflammation; myeloperoxidase; proteinase 3; vasculitis

The first 10% of the full text of this article appears below.

The standard epidemiologic approach to complex diseases tracks down differences in incidence and prevalence rates between distinct populations. Thereby, the potential impact of genetic susceptibility and/or environmental factors will be elucidated and can be dissected on the molecular biologic level in further studies. In this journal issue of Nephrology Dialysis Transplantation, Watts et al. [1] report on the incidence of renal vasculitis in a population from the Norwich area, UK. The authors compared these data on renal involvement in the three anti-neutrophil cytoplasmic autoantibody (ANCA)-associated vasculitides (AAV) Wegener's granulomatosis (WG), microscopic polyangiitis (MPA) and Churg Strauss syndrome (CSS), to recently published incidence rates of a Japanese population [1,2]. The overall . . . [Full Text of this Article]


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