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Nephrology Dialysis Transplantation 2008 23(11):3382-3385; doi:10.1093/ndt/gfn550
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© The Author [2008]. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved. For Permissions, please e-mail: journals.permissions@oxfordjournals.org


Salt intake and cardiovascular disease

Nephrol Dial Transplant 2008; doi:10.1093/ndt/gfn550

Feng J. He and Graham A. MacGregor

Blood Pressure Unit, Cardiac and Vascular Sciences, St George's, University of London, London, UK

Correspondence and offprint requests to: Graham A. MacGregor, Blood Pressure Unit, Cardiac and Vascular Sciences, St George’s, University of London, Cranmer Terrace, London SW17 0RE, UK. Tel: +44-20-8725-2989; Fax: +44-20-8725-2959; E-mail: g.macgreg@sgul.ac.uk

Keywords: blood pressure; cardiovascular disease; salt intake

The first 150 words of the full text of this article appear below.

McCarron, in his review on salt [1], makes some serious and misleading mistakes. For example, he claimed that, in two of our own randomized double-blind crossover trials of modest salt restriction [2,3], we restricted potassium intake. This is incorrect and can be ascertained from a superficial reading of both papers. The average potassium intake on the individuals’ usual diet, as measured by 24-h urinary potassium excretion, was the same as that for the UK population. Secondly, there was no significant change in 24-h urinary potassium with a reduction in salt intake during the run-in period while individuals were on a reduced salt diet. Thirdly, not like McCarron implied, participants did not change their diet during the randomized crossover phase of the studies. Instead they took slow sodium and placebo tablets in a randomized double-blind crossover manner to achieve a difference in salt intake. There was no . . . [Full Text of this Article]

David A. McCarron

President, Academic Network, Executive Director, Shaping America's Youth, 120 NW 9th Avenue, Suite 216, Portland, OR 97209-3326, USA. E-mail: dmccarron@academicnetwork.com


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