Nephrol Dial Transplant (2004) 19: 774-777
Nephrol Dial Transplant Vol. 19 No. 4 © ERA-EDTA 2004; all rights reserved
Editorial Comment
Trends in the prevalence of primary aldosteronism, aldosterone-producing adenomas, and surgically correctable aldosterone-dependent hypertension
1Hypertension Unit and 2Department of Medical Informatics, Hôpital Européen Georges Pompidou, Paris, France
Correspondence and offprint requests to: Dr P. F. Plouin, Hypertension Unit, Hôpital Européen Georges Pompidou, 20 rue Leblanc, 75908 Paris cedex 15, France. Email: pierre-francois.plouin@egp.ap-hop-paris.fr
Keywords: aldosterone; hypertension; secondary; hyperaldosteronism; renin
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Introduction |
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Although primary aldosteronism (PA) has long been considered a rare cause of hypertension, recent reports suggest that the prevalence of PA among hypertensive patients may exceed 10% [1,2]. An actual increase in the true prevalence of PA is unlikely [3,4], but diagnostic advances may result in a more frequent and effective screening for the condition. Screening for PA is no longer limited to patients with hypokalaemia [1–9]. Using the aldosterone to renin ratio (ARR) is a more convenient screening test than separate determinations of plasma renin activity (PRA) and urinary aldosterone excretion [1–6,8,10,11] and, according to some [1–6,10] but not all [11–14] reports, is less influenced by antihypertensive medication.
The diagnosis of PA is not synonymous with the
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Primary aldosteronism in a clinical perspective |
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Primary aldosteronism subtypes |
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Primary aldosteronism, aldosterone-producing adenoma, and cure rate following surgery |
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A pragmatic approach to primary aldosteronism |
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