Skip Navigation

This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (10)
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Schindler, R.
Right arrow Articles by Frei, U.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Schindler, R.
Right arrow Articles by Frei, U.
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Nephrol Dial Transplant (2002) 17: 17-19
© 2002 European Renal Association-European Dialysis and Transplant Association


Editorial Comments

Influencing the inflammatory response of haemodialysis patients by cytokine elimination using large-pore membranes

Ralf Schindler, Raimund Senf and Ulrich Frei

Department of Nephrology and Internal Intensive Care Medicine, Charité-Campus Virchow-Klinikum, Humboldt University of Berlin, Berlin, Germany

Keywords: C-reactive protein; cardiovascular risk; inflammatory mediators

Inflammatory mediators and cardiovascular risk

Over recent years an association between cardiovascular events and inflammation has been reported by several groups and this now finally appears to be well established. Baseline levels of C-reactive protein (CRP) in apparently healthy individuals represent an independent risk factor for cardiovascular events including myocardial infarction and peripheral artery disease [1–3]. Moreover, the rise in CRP after myocardial infarction, or during unstable angina pectoris, predicts outcome [4]. It has been suggested that the inflammatory process may not merely be an epiphenomenon but rather a pathogenetical factor in the genesis of atherosclerosis [5].

Cardiovascular mortality is greatly increased in haemodialysis patients. For instance, the risk of dying from cardiovascular causes is 100-fold . . . [Full Text of this Article]

Removal of mediators by extracorporeal treatment

Need for prospective studies

Notes

References


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
J. Am. Soc. Nephrol.Home page
E. G. Lowrie
Does a Statistical Method Suggest a New Pathobiology for Hemodialysis Patients?
J. Am. Soc. Nephrol., September 1, 2009; 20(9): 1867 - 1869.
[Full Text] [PDF]


Home page
Postgrad. Med. J.Home page
A Power, N Duncan, and C Goodlad
Advances and innovations in dialysis in the 21st century
Postgrad. Med. J., February 1, 2009; 85(1000): 102 - 107.
[Abstract] [Full Text] [PDF]


Home page
Crit Care NurseHome page
S. Dirkes and K. Hodge
Continuous Renal Replacement Therapy in the Adult Intensive Care Unit: History and Current Trends
Crit. Care Nurse, April 1, 2007; 27(2): 61 - 80.
[Full Text] [PDF]


Home page
J Bone Joint Surg BrHome page
A. Grubl, M. Weissinger, W. Brodner, A. Gleiss, A. Giurea, M. Gruber, G. Poll, V. Meisinger, F. Gottsauner-Wolf, and R. Kotz
Serum aluminium and cobalt levels after ceramic-on-ceramic and metal-on-metal total hip replacement
J Bone Joint Surg Br, August 1, 2006; 88-B(8): 1003 - 1005.
[Abstract] [Full Text] [PDF]


Home page
Nephrol Dial TransplantHome page
M. P. C. Grooteman and M. J. Nube
Impact of the type of dialyser on the clinical outcome in chronic haemodialysis patients: does it really matter?
Nephrol. Dial. Transplant., December 1, 2004; 19(12): 2965 - 2970.
[Full Text] [PDF]