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Nephrol Dial Transplant (2001) 16: 1978-1982
© 2001 European Renal Association-European Dialysis and Transplant Association

Editorial Comments

Glycaemic control and graft loss following renal transplantation

Merlin C. Thomas, Timothy H. Mathew and Graeme R. Russ

Renal Unit, The Queen Elizabeth Hospital, Adelaide, South Australia

Keywords: diabetes; glycaemic; rejection; transplantation

Introduction

Diabetes is the single most common reason for end-stage renal disease (ESRD) in the Western world. Currently, one-quarter of all renal transplant patients and almost half of all patients entering renal replacement programs have diabetes [1]. In addition, many patients without diabetic nephropathy show glucose intolerance and manifest hyperglycaemia following transplantation. We have recently reported the development of significant fasting hyperglycaemia (>8.0 mmol/l) immediately following transplant surgery in 73% of patients without diabetes [2]. Moreover, a majority of patients continue to be hyperglycaemic long after surgery. Careful attention to glycaemic control is therefore important for most patients undergoing renal transplantation because, in addition to reductions in post-operative infection [3] and prevention of the vascular complications of diabetes, there is evidence that glycaemic control also contributes to the main causes of allograft loss.

Acute rejection

Patients with diabetes have an increased incidence of acute rejection following renal . . . [Full Text of this Article]

Delayed graft function

Chronic allograft dysfunction

Disease recurrence

Glycaemic control before and after transplantation

Conclusions

Acknowledgments

Notes

References


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