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Nephrol Dial Transplant (2000) 15: 751-753
© 2000 European Renal Association-European Dialysis and Transplant Association

Editorial Comments

Renal failure and hantavirus infection in Europe

Michael Faulde1,, Dirck Sobe1, Peter Kimmig2 and Jerrold Scharninghausen3

1 Central Institute of the Armed Forces Medical Service, Koblenz, Germany, 2 Landesgesundheitsamt Baden-Württemberg, Stuttgart, Germany and 3 Department of Wildlife and Fisheries Sciences, Texas A&M University, College Station, Texas, USA

Introduction

Hantaviruses, the causative agents for haemorrhagic fever with renal syndrome (HFRS) and hantavirus pulmonary syndrome (HPS), are serologically related viruses of the family Bunyaviridae, and have a worldwide distribution. Unlike other Bunyaviruses, hantaviruses are not known to be transmitted by an arthropod vector. The natural hosts of these viruses are small mammals. Hantavirus virions are excreted from infected rodents via saliva, urine, and faeces. Humans may become infected through inhalation of aerosols of dried excreta, inoculation through the conjunctiva, or entry through broken skin or rodent bites [1].

Human hantaviral infections are not new. During the 1950s, hantavirus carried by the striped field mouse, Apodemus agrarius, caused approximately 3000 cases of Korean haemorrhagic fever among United Nations troops participating in the Korean conflict. The aetiologic agent, defined as Hantaan virus, was first isolated in 1977 from the rodent reservoir [2]. Since then, over 20 other . . . [Full Text of this Article]

Pathogenesis and clinical outcome of haemorrhagic fever with renal syndrome and nephropathia epidemica

Diagnosis and treatment

Epidemiological aspects and transmission modes in Europe

Conclusion

Notes

References


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