Nephrol Dial Transplant (1999) 14: 2803-2805
© 1999 European Renal Association-European Dialysis and Transplant Association
Editorial Comments
Not all left ventricular hypertrophy is created equal
Department of Medicine IV/Nephrology, University of Erlangen-Nürnberg, Germany
Correspondence and offprint requests to: Prof. Dr Roland E. Schmieder, Medizinische Klinik IV/Nephrologie, Universität Erlangen-Nürnberg, Breslauer Str. 201, D-90471 Nürnberg, Germany.
Keywords: arterial hypertension; chronic renal failure; left ventricular hypertrophy
Epidemiology of left ventricular hypertrophy
Left ventricular (LV) hypertrophy is a frequent finding in a population with established systemic hypertension with an echocardiographically determined prevalance of up to 48% depending on the definition of the upper normal limit of LV mass. LV hypertrophy is primarily a compensatory mechanism in response to the increased workload imposed on the heart in hypertensive subjects. However, LV hypertrophy represents a major risk factor with respect to cardiovascular morbidity and mortality in primary and secondary arterial hypertension and in end-stage renal disease (ESRD) [1–3]. The increased risk is attributable to several sequelae of LV hypertrophy such as an impaired diastolic filling of the LV cavity, one of the earliest negative consequences of hypertensive heart disease, or an impaired systolic function which both ultimately lead to clinical signs of
Pathophysiology of LV hypertrophy
Geometry of LV hypertrophy
LV hypertrophy in ESRD
LV geometry in ESRD
Therapy of LV hypertrophy in ESRD
References
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