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NDT Advance Access originally published online on August 17, 2004
Nephrology Dialysis Transplantation 2004 19(10):2622-2629; doi:10.1093/ndt/gfh453
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Nephrol Dial Transplant Vol. 19 No. 10 © ERA-EDTA 2004; all rights reserved


Original Article

Impact of chronic allograft nephropathy and subsequent modifications of immunosuppressive therapy on late graft outcomes in renal transplantation

Giuseppe Montagnino1, Giovanni Banfi1, Maria Rosaria Campise1, Patrizia Passerini1, Adriana Aroldi1, Bruno Mario Cesana2 and Claudio Ponticelli1

1 Divisione di Nefrologia e Dialisi, Ospedale Maggiore di Milano, IRCCS, Via Commenda 15 and 2 Laboratorio Epidemiologico, Ospedale Maggiore di Milano, IRCCS, Via F. Sforza 28, 20122 Milan, Italy

Correspondence and offprint requests to: Giuseppe Montagnino, Divisione di Nefrologia e Dialisi, Ospedale Maggiore di Milano, IRCCS, Via Commenda 15, 20122 Milan, Italy. Email: monta{at}policlinico.mi.it

Background. Chronic allograft nephropathy (CAN) is the leading cause of organ failure in renal transplant recipients. We retrospectively evaluated the impact of varying immunosuppression in CAN patients on long-term graft survival.

Methods. We retrospectively analysed 158 cyclosporin (CsA)-treated renal transplant recipients with biopsy-proven CAN with follow-up of >1 year. Immunosuppression remained unchanged in 75 (NOVAR) and was modified in 83 patients (VAR). In 36.1% of VAR patients, it was increased; in 63.8%, the addition of other immunosuppressants was associated with a 20% reduction in or withdrawal of CsA. A regression model, for creatinine clearance (CrCl) slope analysis after therapy variation, and Cox's analysis were applied.

Results. In VAR patients, two-phase regression did not show a correlation between the inflection point in the CrCl slope and treatment variation. Changing immunosuppression gave a borderline advantage in long-term graft survival compared with NOVAR (P = 0.088). In univariate analysis, severe histological lesions, proteinuria >0.5 g/day and CrCl <25 ml/min at biopsy correlated with poor graft outcome (P = 0.0009). In multivariate analysis, only proteinuria and low CrCl remained significative. Stratifying histological lesions in relation to therapy variation showed that severe lesions significantly decreased survival in both VAR and NOVAR groups; however, the highly negative impact of severe lesions in NOVAR patients on graft survival [relative risk (RR) 3.602] was reduced in VAR patients (RR 1.951), with a 10 year graft survival since biopsy of 0.16 vs 0.34 (P = 0.0001).

Conclusions. In transplant patients with CAN, variation of immunosuppression can reduce the negative impact of severe chronic lesions.

Keywords: anti-rejection therapy variation; chronic allograft nephropathy; histology of chronic allograft nephropathy; outcome assessment


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